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While metastatic breast cancer MBC remains incurable, a vast array of active therapeutic agents has provided the opportunity for long-term disease control while maintaining quality of life and physical function. Optimal management of MBC balances a multitude of factors, including a woman's performance status, social support, symptoms, disease burden, prior therapies, and surrogates for tumor biology. Choosing the most appropriate initial therapy and subsequent sequence of treatments demands flexibility as goals and patient preferences may change. Knowledge of the estrogen receptor ERprogesterone receptor PRand Her2 receptor status of the metastatic tumor has become critical to determining the optimal treatment strategy in the metastatic setting as targeted therapeutic approaches are developed.
Gregory D. Leonard, Sandra M. Laboratory and patient data suggest that DCIS is a precursor lesion for invasive cancer.
Breast Surgery. Ananthakrishnan, Director of Breast Surgery, is a fellowship-trained breast surgeon whose practice is dedicated to the treatment of breast disease. She has extensive experience, previously serving on the faculty of the Cleveland Clinic and Columbia University College of Phy
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Cancer mortality and survival rates are much poorer for Black patients than for White patients. We argue that Black—White treatment disparities are a major reason for these disparities. We examine three specific kinds of Black—White treatment disparities: disparities in information exchange in oncology interactions, disparities in the treatment of breast cancer, and disparities in the treatment of clinically localized prostate cancer.
Globally, breast cancer is the most common cancer in women. Heterozygous germ-line mutations in either the BRCA1 or BRCA2 tumour suppressor genes are the most common genetic cause of familial breast cancer and were identified as breast cancer susceptibility genes in the s through linkage analysis in families with the disease Futreal et al. Furthermore, in addition to germ-line mutations in these genes, somatic BRCA1 and BRCA2 mutations are also found in breast, prostate, ovarian and pancreatic cancers, as is somatic hypermethylation of the BRCA1 gene promoter. This triple-negative phenotype precludes the use of targeted oestrogen receptor-based or ERBB2-specific therapies, and in general, BRCA1 mutant breast cancers are treated with traditional genotoxic chemotherapy agents.